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By John McTaggart Ellis McTaggart

Крупнейший представитель британского неогегельянства Дж. Э. Мак-Таггарт анализирует «Науку логики» Гегеля, исследуя диалектику перехода Бытия в Абсолютную Идею. Рассматриваются ключевые понятия гегелевской логики – качество, количество, мера, сущность и явление, действительность, субъект, объект, идея.
Для философов и историков философии.

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Nonhomologous end joining (NHEJ) joins DNA ends with minimal homology and operates in all cell cycle phases. Normally, this is done with a low error rate but a variant backup pathway is more error prone leading to increased rate of mutation (13). The other major pathway, homologous recombination (HR), is essentially error free. It requires a second intact copy of the gene and thus operates in the G2 phase of the cell cycle after DNA has been replicated. Although most DNA damage is correctly repaired, misrepair may lead to cell death or mutation.

MIN, on the other hand, is associated with an increase in the mutation rate at the nucleotide level caused by a defect in the MMR system and appears to be mutually exclusive with CIN (19). Types of genes involved in carcinogenesis Mutations and changes in gene expression can contribute to carcinogenesis at different levels. Thus constant stimulation of proliferation by growth factors or cytokines, such as the epidermal growth factor (EGF) may be caused by activation, gene amplification or autocrine loops.

If a person has inherited a mutant allele of a tumour suppressor gene from one parent, the cells will be heterozygote for that gene but the other allele is usually sufficient for normal function. However, if the other allele is mutated or lost later, the tumour suppressor function is lost. This is an example of loss of heterozygosity (LOH) indicative of genomic instability and mutations involved in malignant transformation. Genomic instability is a common feature of cancer cells that may increase the probability of these steps occurring.

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