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Furthermore, various oncogenes can be studied in cell proliferation and expression of specific extracellular matrix components in chondrocytes. The effects of myc and scr oncogenes have been studied in this way by Alema et al. (1985). The study showed that scr oncogene suppressed the synthesis of type II collagen and cartilage-specific sulfated proteoglycan, whereas myc oncogene did not suppress the synthesis of these normal differentiated cell products. 38 M. ADOLPHE These basic experiments could be extended to the use of genetic approaches for the study of the effects of drug exposure.

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Tissue React. 3, 145-150. , and Lechat, P. (1983). Cell Tissue Kinet. 16, 531-537. , H a r t m a n n , D. , and Adolphe, M. (1984). Biol. Cell. 51, 307-314. , Guillosson, J. , and Adolphe, M. (1985). Biol. Cell. 55, 5a. Saarni, H. (1977). Biochem. Pharmacol. 26, 1961-1966. Sachs, B. , Goldberg, V. , Moskowitz, R. , and Malemud, C. J. (1982). J. Cell. Physiol. 112, 5 1 - 5 9 . Sapolsky, A. , Malemud, C. , Norby, D. , Moskowitz, R. , and Howell, D. S. (1981). Biochim. Biophys. Ada 658, 138-147. Saxe, A.

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